KIBRA (WWC1) Is a Metastasis Suppressor Gene Affected by Chromosome 5q Loss in Triple-Negative Breast Cancer - ScienceDirect
Article | KIBRA (WWC1) Is a Metastasis Suppressor Gene Affected by Chromosome 5q Loss in Triple-Negative Breast Cancer | ID: 3197xr10h | eScholarship@McGill
KIBRA is associated with accelerated cognitive decline and hippocampal atrophy in APOE ε4-positive cognitively normal adults with high Aβ-amyloid burden | Scientific Reports
Frontiers | The Neuroprotection of KIBRA in Promoting Neuron Survival and Against Amyloid β-Induced Apoptosis
MiR-21 targeted the hub gene KIBRA. (A) The heatmap of 16... | Download Scientific Diagram
KIBRA: In the brain and beyond. - Abstract - Europe PMC
KIBRA gene methylation is associated with unfavorable biological prognostic parameters in chronic lymphocytic leukemia: Epigenetics: Vol 7, No 3
The memory gene KIBRA is a bidirectional regulator of synaptic and structural plasticity in the adult brain | Semantic Scholar
Frontiers | KIBRA: a new gateway to learning and memory?
Structural features of the human KIBRA protein. Shown are the... | Download Scientific Diagram
WWC1 Gene - GeneCards | KIBRA Protein | KIBRA Antibody
KIBRA: In the brain and beyond - ScienceDirect
KIBRA: In the brain and beyond. - Abstract - Europe PMC
JCI Insight - KIBRA upregulation increases susceptibility to podocyte injury and glomerular disease progression
Common exonic missense variants in the C2 domain of the human KIBRA protein modify lipid binding and cognitive performance | Translational Psychiatry
Impact of Common KIBRA Allele on Human Cognitive Functions | Neuropsychopharmacology
Loss of KIBRA function activates EGFR signaling by inducing AREG | Oncotarget
Kibra Modulates Learning and Memory via Binding to Dendrin - ScienceDirect
KIBRA-PKCγ signaling pathway modulates memory performance in mice and humans | bioRxiv
KIBRA regulates amyloid β metabolism by controlling extracellular vesicles secretion - eBioMedicine
KIBRA Team Up with Partners to Promote Breast Cancer Metastasis | SpringerLink
